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16 Although the exact pathophysiology of delirium remains poorly understood, some of the leading hypotheses for the pathogenesis for delirium viagra 100mg tablets 4g on the role of neurotransmission, inflammation, and chronic stress. 12,13 There exists ample evidence for cholinergic deficiency and dopaminergic excess in delirium. The fact that anticholinergic and dopaminergic drugs precipitate delirium, and cholinergiccholinesterase inhibitors and dopamine antagonists relieve delirium speaks for this hypothesis. 12,13 The involvement of other neurotransmiters such as norepinephrine, g-aminobutyric acid, glutamate, and serotonin is also implicated. Inflammation increases the permeability of the blood-brain barrier. 12,16 Stress activates the sympathetic nervous system and the hypothalamic-pituitary-adrenocortical system. These changes in neurotransmission could be the cause for the psychotic symptoms in delirium.

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Tardive dyskinesia rates with atypical antipsychotics in adults: prevalence and incidence. J Clin Psychiatry. 2004;65(suppl 9):16-20. Adler LA. Vitamin E treatment for tardive dyskinesia. Veterans Affairs Cooperative Study 394 Study Group.

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