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NO achieves its smooth-muscle relaxation effect by increasing the formation of cGMP, which in turn mobilizes Ca, thereby invoking mechanisms for smooth-muscle relaxation. cGMP is hydrolyzed to 5146;GMP by the enzyme phosphodiesterase (PDE). Inhibition of PDE by sildenafil potentiates the activity of NO by preserving higher corporal levels of the cyclic nucleotide. Thus, sildenafil potentiates the action of natural stimuli to arousal. It does not directly increase cGMP. Prostacyclin, on the other hand, is synthesized from arachidonate and induces smooth-muscle relaxation by activating cAMP.

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